82-Year-Old Woman With Dementia Gets Memory Back After Changing Her Diet

After changing her diet, an 82-year-old woman who suffered from dementia and was not able to recognize her own son has miraculously restored her memory.

When her condition became too severe, her son Mark Hatzer left her in the hospital, and almost came to terms that he has lost her. Her state was really grave, and due to the memory loss and the loss of parts of her mind, she even phoned the police once to accuse the nurse who was caring for her of kidnap.

However, a change of her diet, which was comprised of high levels of walnuts and blueberries, drastically improved her condition.

The mother and son both comprised a diet after they saw that her medication was not enough to treat her dementia. Through research, they found that people in the Mediterranean countries have lower rates of dementia, so they copied their eating habits.

She started consuming healthy foods like oats, kale, broccoli, spinach, sweet potatoes, green tea, dark chocolate, and sunflower seeds, all of which are known to improve brain health.

Mark, whose brother Brent also died in 1977, said that his mother was in a hotel, but the worst she had ever been. She didn’t recognize him and phoned the police, believing she was kidnapped. 

It was devastating, since they were very close, especially after his father and brother died. He felt alone and despaired, with no one to turn to.

Therefore, when she left the hospital, they decided to try an alternative treatment, instead of prescribed medication.  He says that Alzheimer’s is very rare in certain countries due to their diets, rich in foods that even resemble the brain in shape, like walnuts, Brazil nuts, as well as fish, blueberries, strawberries.

He and his mom also did some cognitive exercises like jigsaw puzzles crosswords and meeting people in social situations, and after a few months, she began remembering things like birthdays and began more engaged and alert. 

Her son says that now, she looks 10 years younger, and if you met her now, you would not know she had gone through all those struggles.  Their story is another proof that under the right conditions, our bodies are highly resilient, and a proper diet can teat various types of diseases.

Additionally, researchers have been warning about the negative effects of aluminum buildup in the brain and its link to dementia and more specifically Alzheimer’s disease, for some time now.

Therefore, it is of high importance to take the needed measures and limit the exposure to this heavy metal as well. 

The Hippocratic Post website published an article named Strong evidence linking Aluminum to Alzheimer’s, in which Professor Exley explained that the aluminum content of brain tissue in late-onset or sporadic Alzheimer’s disease is significantly higher than is found in age-matched controls.

This means that people who develop Alzheimer’s disease in their late 60s and older build up more aluminum in their brain tissue than people of the same age without the disease.

Even elevated levels of aluminum have been found in the brains of people diagnosed with an early-onset form of sporadic Alzheimer’s disease, who have experienced an unusually high exposure to aluminum.  Therefore, it has a much earlier age of onset in people who have been exposed to unusually high levels of aluminum in their everyday lives.

In December 2016, the Journal of Trace Elements in Medicine and Biology published another study titled: Aluminum in brain tissue in familial Alzheimer’s disease, which is highly important, as it is the first time that scientists have measured the aluminum level in the brain tissue of individuals diagnosed with familial Alzheimer’s disease.

The paper showed that the aluminum concentrations found in brain tissue donated by individuals who died with a diagnosis of familial AD were the highest level ever measured in human brain tissue.

Professor Exley also wrote that some of the highest levels of aluminum ever measured in human brain tissue are found in people who have died with a diagnosis of familial Alzheimer’s disease. Their levels of aluminum in the brain were similar to those recorded in people who died of an aluminum-induced encephalopathy while undergoing renal dialysis.

Familial Alzheimer’s disease is an early-onset form of the disease, with symptoms occurring as early as 30 or 40 years of age. It is extremely rare, 2-3% of all cases of Alzheimer’s disease.

It is based on genetic mutations linked to a protein called amyloid-beta, which has been heavily linked with the cause of all Alzheimer’s causes. These people produce more amyloid beta and the onset of the symptoms of Alzheimer’s disease occur much earlier in life.

Furthermore, Dr. Joseph Mercola speaks about other potential factors that contribute to the development of dementia and Alzheimer’s, which we need to consider. Namely, studies have discovered that high-carb diets raise the risk of dementia by 89 percent, while high-fat diets reduce it by 44 percent.

Alzheimer’s disease is linked to insulin resistance; even mild elevation of blood sugar is related to higher dementia risk. Diabetes and heart disease raise the risk, and both are rooted in insulin resistance.

The link between high-sugar diets and Alzheimer’s was again emphasized in a longitudinal study published in the journal Diabetologia in January 2018. Researchers followed about 5,190 individuals over a decade and found that the higher an individual’s blood sugar, the faster their rate of cognitive decline.

Additionally, even the bodies of Type 1 diabetics do not produce insulin at all, they are still at increased risk for Alzheimer’s. This paradox was investigated in 2016 by Melissa Schilling, a professor at New York University.

She claims that it is a result of the insulin-degrading enzyme, a product of insulin that breaks down both insulin and amyloid proteins in the brain, which accumulate and lead to Alzheimer’s disease.

People with insufficient insulin, like those whose bodies cannot produce it properly due to diabetes, going to make enough of this enzyme to break up those brain clumps.

Also, in individuals who use insulin to treat their diabetes and end up with an insulin surplus, most of this enzyme gets used up breaking that insulin down, leaving not enough enzyme to fight the amyloid brain clumps. Schilling claims that this can also happen in the case of prediabetes.

Therefore, the more the awareness about the causes of neurodegenerative brain disorders grows, the more we can do to treat and prevent them altogether.